Category Archives: Symptoms

Wheelchair Accessible Vehicles: Q&A

Wheelchair
Accessible Vans

Rear entry Vs. Side entry
Buying New Vs. Buying Used
Manual Ramp Vs. Powered Ramp
Honda Vs. Dodge/Chrysler Vs. Toyota Vs. Ford
Certified Mobility Dealer Vs. Car Dealer Vs. Buying online
What do you need to know to get maximum benefit for minimum expense?

Good information is the key to saving money and getting the most value for the dollar when making a big-ticket purchase like a wheelchair-accessible vehicle.

With that in mind, Seek out and find experts who truly care. Here are some answers to common questions about adaptive mobility equipment.

Can I just go to a car dealer down the street or do I need a certified mobility dealer?

Certified mobility dealers will help you buy the right vehicle and adaptive mobility equipment to meet your needs now and in the future. Future planning is especially important for people with muscle diseases that get progressively worse over time.

“Technology has improved tremendously over the years so there are numerous products available. Our goal is to help people find the right equipment that best fits their needs,” says Jim Sanders, president of Automotive Innovations based in Bridgewater, MA for over 25 years.

“Many times, consumers will go to a car dealer and buy a vehicle that can’t be modified or one that doesn’t fit their needs. And once you buy a vehicle, normally it’s very difficult to return.”

The National Mobility Equipment Dealers Association (NMEDA), a nonprofit organization that provides consumer guidance and ensures quality and professionalism in the manufacturing and installation of mobility equipment. Members include mobility equipment dealers, manufacturers, driver rehabilitation specialists and other professionals.

NMEDA member-dealers must follow the safety standards established by the National Highway Traffic Safety Administration (NHTSA), in addition to NMEDA’s own stringent guidelines.

Some dealers choose to enroll in NMEDA’s Quality Assurance Program (QAP), which requires them to adhere to national motor vehicle safety standards, and use proven quality control practices to yield the highest level of performance and safety. Automotive Innovations was the First Mobility Dealer in Massachusetts to enroll and exceed the safety standards.

“The QAP dealer is audited by an outside engineering firm to verify that technicians have been trained and that the dealer has insurance and make sure the facility is ADA-compliant,” which means the QAP dealer is going above and beyond.

 

Can I get a better price if I buy online rather than from a dealer?

As with any online shopping, the warning “buyer beware” rings true. Buying online without trying out different vehicles with different conversions can be a costly mistake. Furthermore there are many grey market converted vans being offered as quality conversions.

Online, you are mostly shopping blind. Typically you will have no idea how the vehicle you need will work for you, even with specific recommendations from a driver evaluator or occupational therapist.

“You definitely shouldn’t buy a wheelchair accessible vehicle online, most online sellers are not qualified Mobility Dealers attempting to assess your needs, they’re just car dealers trying to sell you something.”

Some online dealers even have questionnaires on their websites to try and give you the idea your getting what you need. But, it will never replace being able to go to a local mobility dealership and try the vans out first hand.

A mobility vehicle is probably the second-largest purchase after a house. You should see it, try it out, and make sure it’s something that will work for you and your family. It’s horrible when people spend so much an a vehicle that will never work for them.

Every vehicle is a little bit different — such as in the dimensions, electrical and fuel systems, or suspension modifications. “If you go online and buy a wheelchair accessible vehicle based on the price, you’re not really looking at the total package.”

While buying online may be able to save you some money up front, it won’t over the long term.

In addition to you missing out on the important local service contact that a mobility equipment dealer provides, these online deals or grey market vans are worth much less when it comes time to trade it in.

 

What are some common mistakes people make when buying a modified vehicle?

Manufacturers and mobility dealers agree that one of the most common — and costly — mistakes is buying the vehicle first and then shopping for the conversion or adaptive mobility equipment. Not all vehicles can be converted.

For example, If you purchase a minivan from a traditional car dealership you can hit a roadblock if it doesn’t meet specific requirements to have the floor lowered for a rear- or side-entry conversion.

 

What are some good questions to ask a dealer or manufacturer?

Although buying a modified vehicle can be “a daunting experience,” says VMI’s Monique McGivney, it also can be “exciting and fun when you walk in armed with good questions and information.”

Prior to getting an assessment from a mobility dealer, evaluate your needs and try answering the following questions:

  • What vehicle will fit in my garage?
  • What kind of parking issues will I encounter where I live?
  • What is the size and weight of my wheelchair?
  • What is my seated height in the wheelchair?
  • How many people will ride in the vehicle?
  • In what part of the vehicle do I want to sit?
  • Will I be able to drive with hand controls?
  • Do I want a full-size van, minivan or alternative vehicle?
  • Do I want manual or power equipment?
  • Will an in-floor ramp or fold-out ramp meet my needs?
  • What is my budget, and do I have access to supplemental funding?

The first question most mobility dealers will ask you is: “What is your seated height in the wheelchair?” From there, the dealer can advise whether a full-size or minivan is appropriate, and what kind of conversion is needed.

Be sure to ask the dealer about the warranty and how the vehicle can be serviced.

Which Make and Model is the best for a handicapped accessible vehicle?

It honestly depends on what you fit into best and what options you prefer.

No two wheelchair accessible vehicles are the same. They vary in size, shape, color, features and design depending on the vehicle’s make and model. The only way to guarantee which is the best vehicle for you is if you come in and try them all out.

For example: The Honda has a little bit more room inside to maneuver a wheelchair than a Dodge, just as a Toyota has a bit more space than a Honda. A Ford offers more headroom than all of the above. But that all depends on the conversion and manufacturer.

Although color and features matter least to us, some find them just as important as fitting into the vehicle. Each Manufacturer offers their own color schemes, which you can look up on their websites. You can also search for what features you would prefer to have.

When you come into our Mobility Center we will help you find the vehicle that best fits you and your family’s needs. If you love the vehicle but not the color or features we can custom order a vehicle for you. That way we know you are buying a vehicle that best fits you and one that you are 100% happy with.

Which is better: rear entry or side entry?

The most important difference between a rear entry and side-entry conversion is that with a rear entry, wheelchair users can’t drive from their wheelchairs nor can they ride in the front passenger seat. From there, the choice comes down to personal preference and budget.

In recent years, because of quality, convenience and cost, there’s been a shift toward side entry vehicles. Rear entry is more of a frugal modification, involves a less of conversion process and is typically a little less expensive than a side-entry conversion.

Many people prefer side entry with an in-floor conversion for many safety reasons additionally  because they can park almost anywhere and not worry deploying the ramp out into traffic. Also, side entry allows the consumer to ride in the passengers front position along with maintain the rear seats in a minivan because the conversion doesn’t affect that area.

Rear entry is harder to get out of compared to a side-entry.

Anyway you look at it side-entry vehicles are more versatile. For example, side entry allows someone with a progressively worsening condition to use the vehicle for a longer period of time. A wheelchair user can start out driving from his or her chair, and then move to several other positions in the vehicle when no longer able to drive.

Side-entry conversions typically are a little more expensive than rear-entry because they’re more intrusive and labor intensive. For example, with a minivan, the entire floor and frame must be removed and replaced with a lowered floor and new frame.


What’s the difference between a fold-out ramp and in-floor ramp?

This decision comes down to safety, aesthetics, convenience and cost.

A fold-out ramp folds up into the vehicle, takes up valuable space in the passengers front area and must be deployed whenever the door is opened.

The in-floor ramp slides under the floor which makes riding in the vehicle safer for anyone seated in the passengers front position or the mid-ship position. There is no obstruction to the doorway so other passengers can enter and exit without deploying the ramp. In-floor ramps are currently only available as a side-entry minivan conversion, but they offer a manual (un-powered) option as well.

In-floor ramps in addition to being safer will generally provide more room in the vehicle because there’s nothing blocking the doorway. The ramp is “out of sight, out of mind” and may last longer because it doesn’t have to be deployed each time the side passenger door opens.

Fold-out ramps generally cost a little less than an in-floor ramp and consumers can select from manual and power versions; a power fold-out ramp still costs less than an in-floor ramp.

If an in-floor ramp system breaks down or the vehicle loses power, VMI’s in-floor ramp systems have a backup system (sure-deploy) that bypasses the vehicle’s battery.

A lot of people just feel more secure knowing there isn’t a fold-out ramp next to them in the event of a accident.

I use a wheelchair, but a van or minivan just isn’t “me.” Are they my only options?

You have other choices.

Lowered-floor conversions with fold-out ramps can be done on the Honda Element, Chrysler PT Cruiser and Toyota Scion. The conversions are small and don’t fit as many people.

Due to them being built on a much smaller scale, the ones we have seen have not been built with the same level of quality as the minivan conversion. Parts availability and repairs have been a problem, some of the companies that converted them are out of business and or have no support for “something they used to build”

If you prefer to keep your standard car rather than purchasing a modified vehicle — and can make the transfer from a wheelchair to a car seat — the answer may be as simple as a set of hand controls or a left foot gas pedal

Turning seats can be used in a wide range of vehicles, from sedans to SUVs and pickup trucks. A way to transport the wheelchair (like a rear lift) also is needed.

The rate at which your symptoms worsen is one thing to consider when looking at turning seats — is it likely you’ll be able to transfer and ride in a car seat for many more years? Also, be sure to check with a mobility dealer to determine if your vehicle can accommodate a turning seat and a wheelchair lift.

Why are modified vehicles so  expensive?

A vehicle conversion can cost consumers upwards of $27,000 —  and that’s just the cost for the conversion, not the vehicle. The total package can run between $45,000 and $80,000 — or more.

Besides the cost of the components, the reason it’s so pricey is that basically there is a lot of work involved to build a quality vehicle.

Modified vehicles from certified manufacturers and dealers must meet NHTSA’s Federal Motor Vehicle Safety Standards (FMVSS). That means all modified vehicles must be properly crash tested. (To learn more, visit www.nhtsa.dot.gov.)

It’s quite a labor-intensive process because of the customization. When you make structural modifications to a vehicle, you have to go through all of the crash testing, and you have to show that the vehicle is compliant again, and those tests are very expensive.

Most of the time lowering the floor in a minivan requires replacing or moving the fuel tank. Once the conversion is finished, the vehicle still has to meet the original requirements for evaporative emissions, in addition to NHTSA requirements.

How can I pay less?

You have  a few options.

You could cut costs by purchasing a pre-owned vehicle with a new conversion, typically saving you around $10,000 to $12,000.

The previous van owner already has absorbed the depreciation hit on a new van, which essentially occurs right after they’ve driven off the dealer’s lot.

Buying used can be beneficial for first-time buyers who want to try out a vehicle for a few years before buying new.

But if you plan to buy used, do some research and make sure the vehicle is structurally sound including the conversion. Ask for a vehicle history (CARFAX) report, and get the vehicle inspected by a mobility dealer to ensure it’s in good shape and was well taken care of.

Another tactic to help save you money is to ask your Certified Mobility Dealer about any rebates or financial aid options that could benefit you.

How do people manage to pay for it?

Many consumers used home equity loans to purchase a vehicle and adaptive equipment.

Many dealers and manufacturers work with lending institutions that offer extended-term financing, including 10-year loans, allowing consumers to make lower, more affordable monthly payments. The downside is that consumers are locked into the vehicle for 10 years, and end up paying more in interest.

If you finance for 10 years, and you’re not going to keep the vehicle for that amount of time, you’re going to lose money when you try to sell or trade it because you haven’t paid off much of the balance.

When you buy a new vehicle, many car manufacturers offer mobility reimbursement programs (up to $1,000) to help offset the cost for the purchase and installation of adaptive equipment.

Caregiver Fatigue Syndrome

Caregiver Fatigue Syndrome

Caregivers walk a very delicate line. They are some of the strongest, most selfless individuals around — doing most anything and everything for their loved ones — but many are exhausted, stressed and overwhelmed trying to balance the rewarding process.

Caregiver stress, aka Caregiver Fatigue Syndrome or Caregiver Burnout, is the byproduct of the countless responsibilities, physical demands, strained time and rollercoaster circumstances that come with the turf. And, since many caregivers consistently put others before themselves, if not addressed, this stress will only snowball.

It’s like the airplane oxygen illustration: Before you help others assemble their masks, you must first ensure you have your own. In this regard, before a caregiver can expect to offer their loved one in a wheelchair or loved one with a disability the best care, they must first take care of themselves.

Any imbalance, and the quality of care for your loved one can decrease while your personal stress and fatigue begin to climb.

Physical Symptoms
Many outsiders don’t realize what it takes to be a caregiver. Unless they’ve been in a similar situation, it’s hard to grasp the physical strain of all the lifts and pulls. Transport has been simplified with the help of mobility vehicles, but there are still the routine transitions from bed to bathroom to dinner and then back to bed. Caregiving is hard work.

If you experience any of the following symptoms, you may have caregiver burnout:
● Decrease in overall energy
● Decrease in immunity (subject to more frequent colds/flus)
● Inconsistent sleep patterns
● Chronic back and/or joint pain
● Weight gains

Emotional Drain
In addition to the uncertainty of medical circumstances and the rollercoaster ride that your loved one may be experiencing, it’s not uncommon for a caregiver to feel alone or assume that nobody understands their situation. Activities and hobbies that once brought you joy may be sidelined to your responsibilities and the constant championing of your loved one might not leave much room for your own pursuits.

You may experience:
● Irritability and frustration
● Difficulty relaxing
● Impatience
● Feelings of hopelessness and helplessness
● Dissatisfaction

Mental Exhaustion
Scheduling, logistics, medications and meals — caregivers are the gatekeepers, chauffeurs, advocates, cooks and everything in between for their loved one. There are too many moving parts to name. It’s no wonder things begin to get mentally draining.

Mental exhaustion may be exhibited through:
● Difficulty concentrating
● Feelings of confusion
● Periods of tunnel vision

No matter the symptoms, caregiver fatigue is a serious concern and you are not alone. Don’t buy into the idea that you’re the only caregiver facing these challenges.

If high-quality care for your loved one tops your list, remember that the best starts with the best you.

Limb-Girdle Muscular Dystrophy

What is limb-girdle muscular dystrophy?
Limb-girdle muscular dystrophy is a term for a group of diseases that cause weakness and wasting of the muscles in the arms and legs. The muscles most affected are those closest to the body (proximal muscles), specifically the muscles of the shoulders, upper arms, pelvic area, and thighs.

The severity, age of onset, and features of limb-girdle muscle dystrophy vary among the many subtypes of this condition and may be inconsistent even within the same family. Signs and symptoms may first appear at any age and generally worsen with time, although in some cases they remain mild.

In the early stages of limb-girdle muscular dystrophy, affected individuals may have an unusual walking gait, such as waddling or walking on the balls of their feet, and may also have difficulty running. They may need to use their arms to press themselves up from a squatting position because of their weak thigh muscles. As the condition progresses, people with limb-girdle muscular dystrophy may eventually require wheelchair assistance.

Muscle wasting may cause changes in posture or in the appearance of the shoulder, back, and arm. In particular, weak shoulder muscles tend to make the shoulder blades (scapulae) “stick out” from the back, a sign known as scapular winging. Affected individuals may also have an abnormally curved lower back (lordosis) or a spine that curves to the side (scoliosis). Some develop joint stiffness (contractures) that can restrict movement in their hips, knees, ankles, or elbows. Overgrowth (hypertrophy) of the calf muscles occurs in some people with limb-girdle muscular dystrophy.

Weakening of the heart muscle (cardiomyopathy) occurs in some forms of limb-girdle muscular dystrophy. Some affected individuals experience mild to severe breathing problems related to the weakness of muscles needed for breathing.

Intelligence is generally unaffected in limb-girdle muscular dystrophy; however, developmental delay and intellectual disability have been reported in rare forms of the disorder.

How common is limb-girdle muscular dystrophy?
It is difficult to determine the prevalence of limb-girdle muscular dystrophy because its features vary and overlap with those of other muscle disorders. Prevalence estimates range from 1 in 14,500 to 1 in 123,000 individuals.

Myasthenia Gravis

What is myasthenia gravis?
Myasthenia gravis is a chronic autoimmune neuromuscular disease characterized by varying degrees of weakness of the skeletal (voluntary) muscles of the body. The name myasthenia gravis, which is Latin and Greek in origin, literally means “grave muscle weakness.” With current therapies, however, most cases of myasthenia gravis are not as “grave” as the name implies. In fact, most individuals with myasthenia gravis have a normal life expectancy.

The hallmark of myasthenia gravis is muscle weakness that increases during periods of activity and improves after periods of rest. Certain muscles such as those that control eye and eyelid movement, facial expression, chewing, talking, and swallowing are often, but not always, involved in the disorder. The muscles that control breathing and neck and limb movements may also be affected.

What causes myasthenia gravis?
Myasthenia gravis is caused by a defect in the transmission of nerve impulses to muscles. It occurs when normal communication between the nerve and muscle is interrupted at the neuromuscular junction—the place where nerve cells connect with the muscles they control. Normally when impulses travel down the nerve, the nerve endings release a neurotransmitter substance called acetylcholine. Acetylcholine travels from the neuromuscular junction and binds to acetylcholine receptors which are activated and generate a muscle contraction.

In myasthenia gravis, antibodies block, alter, or destroy the receptors for acetylcholine at the neuromuscular junction, which prevents the muscle contraction from occurring. These antibodies are produced by the body’s own immune system. Myasthenia gravis is an autoimmune disease because the immune system—which normally protects the body from foreign organisms—mistakenly attacks itself.

What is the role of the thymus gland in myasthenia gravis?
The thymus gland, which lies in the chest area beneath the breastbone, plays an important role in the development of the immune system in early life. Its cells form a part of the body’s normal immune system. The gland is somewhat large in infants, grows gradually until puberty, and then gets smaller and is replaced by fat with age. In adults with myasthenia gravis, the thymus gland remains large and is abnormal. It contains certain clusters of immune cells indicative of lymphoid hyperplasia—a condition usually found only in the spleen and lymph nodes during an active immune response. Some individuals with myasthenia gravis develop thymomas (tumors of the thymus gland). Thymomas are generally benign, but they can become malignant.

The relationship between the thymus gland and myasthenia gravis is not yet fully understood. Scientists believe the thymus gland may give incorrect instructions to developing immune cells, ultimately resulting in autoimmunity and the production of the acetylcholine receptor antibodies, thereby setting the stage for the attack on neuromuscular transmission.

What are the symptoms of myasthenia gravis?
Although myasthenia gravis may affect any voluntary muscle, muscles that control eye and eyelid movement, facial expression, and swallowing are most frequently affected. The onset of the disorder may be sudden and symptoms often are not immediately recognized as myasthenia gravis.

In most cases, the first noticeable symptom is weakness of the eye muscles. In others, difficulty in swallowing and slurred speech may be the first signs. The degree of muscle weakness involved in myasthenia gravis varies greatly among individuals, ranging from a localized form limited to eye muscles (ocular myasthenia), to a severe or generalized form in which many muscles—sometimes including those that control breathing—are affected. Symptoms, which vary in type and severity, may include a drooping of one or both eyelids (ptosis), blurred or double vision (diplopia) due to weakness of the muscles that control eye movements, unstable or waddling gait, a change in facial expression, difficulty in swallowing, shortness of breath, impaired speech (dysarthria), and weakness is the arms, hands, fingers, legs, and neck.

Who gets myasthenia gravis?
Myasthenia gravis occurs in all ethnic groups and both genders. It most commonly affects young adult women (under 40) and older men (over 60), but it can occur at any age.

In neonatal myasthenia, the fetus may acquire immune proteins (antibodies) from a mother affected with myasthenia gravis. Generally, cases of neonatal myasthenia gravis are temporary and the child’s symptoms usually disappear within 2-3 months after birth. Other children develop myasthenia gravis indistinguishable from adults. Myasthenia gravis in juveniles is uncommon.

Myasthenia gravis is not directly inherited nor is it contagious. Occasionally, the disease may occur in more than one member of the same family.

Rarely, children may show signs of congenital myasthenia or congenital myasthenic syndrome. These are not autoimmune disorders, but are caused by defective genes that produce abnormal proteins instead of those which normally would produce acetylcholine, acetylcholinesterase (the enzyme that breaks down acetylcholine), or the acetylcholine receptor and other proteins present along the muscle membrane.

How is myasthenia gravis diagnosed?
Because weakness is a common symptom of many other disorders, the diagnosis of myasthenia gravis is often missed or delayed (sometimes up to two years) in people who experience mild weakness or in those individuals whose weakness is restricted to only a few muscles.

The first steps of diagnosing myasthenia gravis include a review of the individual’s medical history, and physical and neurological examinations. The physician looks for impairment of eye movements or muscle weakness without any changes in the individual’s ability to feel things. If the doctor suspects myasthenia gravis, several tests are available to confirm the diagnosis.

A special blood test can detect the presence of immune molecules or acetylcholine receptor antibodies. Most patients with myasthenia gravis have abnormally elevated levels of these antibodies. Recently, a second antibody—called the anti-MuSK antibody—has been found in about 30 to 40 percent of individuals with myasthenia gravis who do not have acetylcholine receptor antibodies. This antibody can also be tested for in the blood. However, neither of these antibodies is present in some individuals with myasthenia gravis, most often in those with ocular myasthenia gravis.

The edrophonium test uses intravenous administration of edrophonium chloride to very briefly relieve weakness in people with myasthenia gravis. The drug blocks the degradation (breakdown) of acetylcholine and temporarily increases the levels of acetylcholine at the neuromuscular junction. Other methods to confirm the diagnosis include a version of nerve conduction study which tests for specific muscle “fatigue” by repetitive nerve stimulation. This test records weakening muscle responses when the nerves are repetitively stimulated by small pulses of electricity. Repetitive stimulation of a nerve during a nerve conduction study may demonstrate gradual decreases of the muscle action potential due to impaired nerve-to-muscle transmission.

Single fiber electromyography (EMG) can also detect impaired nerve-to-muscle transmission. EMG measures the electrical potential of muscle cells when single muscle fibers are stimulated by electrical impulses. Muscle fibers in myasthenia gravis, as well as other neuromuscular disorders, do not respond as well to repeated electrical stimulation compared to muscles from normal individuals.

Diagnostic imaging of the chest, using computed tomography (CT) or magnetic resonance imaging (MRI), may be used to identify the presence of a thymoma.

Pulmonary function testing, which measures breathing strength, helps to predict whether respiration may fail and lead to a myasthenic crisis.

How is myasthenia gravis treated?
Today, myasthenia gravis can generally be controlled. There are several therapies available to help reduce and improve muscle weakness. Medications used to treat the disorder include anticholinesterase agents such as neostigmine and pyridostigmine, which help improve neuromuscular transmission and increase muscle strength. Immunosuppressive drugs such as prednisone, azathioprine, cyclosporin, mycophenolate mofetil, and tacrolimus may also be used. These medications improve muscle strength by suppressing the production of abnormal antibodies. Their use must be carefully monitored by a physician because they may cause major side effects.

Thymectomy, the surgical removal of the thymus gland (which often is abnormal in individuals with myasthenia gravis), reduces symptoms in some individuals without thymoma and may cure some people, possibly by re-balancing the immune system. Thymectomy is recommended for individuals with thymoma. Other therapies used to treat myasthenia gravis include plasmapheresis, a procedure in which serum containing the abnormal antibodies is removed from the blood while cells are replaced, and high-dose intravenous immune globulin, which temporarily modifies the immune system by infusing antibodies from donated blood. These therapies may be used to help individuals during especially difficult periods of weakness. A neurologist will determine which treatment option is best for each individual depending on the severity of the weakness, which muscles are affected, and the individual’s age and other associated medical problems.

What are myasthenic crises?
A myasthenic crisis occurs when the muscles that control breathing weaken to the point that ventilation is inadequate, creating a medical emergency and requiring a respirator for assisted ventilation. In individuals whose respiratory muscles are weak, crises—which generally call for immediate medical attention—may be triggered by infection, fever, or an adverse reaction to medication.

What is the prognosis?
With treatment, most individuals with myasthenia can significantly improve their muscle weakness and lead normal or nearly normal lives. Some cases of myasthenia gravis may go into remission—either temporarily or permanently—and muscle weakness may disappear completely so that medications can be discontinued. Stable, long-lasting complete remissions are the goal of thymectomy and may occur in about 50 percent of individuals who undergo this procedure. In a few cases, the severe weakness of myasthenia gravis may cause respiratory failure, which requires immediate emergency medical care.

World Alzheimer’s Day

World Alzheimer’s Day, September 21st of each year, is a day on which Alzheimer’s organizations around the world concentrate their efforts on raising awareness about Alzheimer’s and dementia. Alzheimer’s disease is the most common form of dementia, a group of disorders that impairs mental functioning.

Every 68 seconds, someone develops Alzheimer’s disease. At current rates, experts believe the number of Americans living with Alzheimer’s will quadruple to as many as 16 million by the year 2050.

Alzheimer’s disease is often called a family disease, because the chronic stress of watching a loved one slowly decline affects everyone. 5.4 million Americans are living with Alzheimer’s. Alzheimer’s disease is the sixth-leading cause of death in the United States and the only cause of death among the top 10 in the United States that cannot be prevented, cured or even slowed. With the increases in life spans and baby boomers coming of age, support for Alzheimer’s research is more critical to our families than ever.